The series of molecular signals generated as a consequence of a fibroblast growth factor receptor binding to one of its physiological ligands and contributing to the progression of the heart over time.

Any process that activates or increases the frequency, rate or extent of phosphatidate phosphatase activity.

The series of molecular signals initiated by the binding of a member of the BMP (bone morphogenetic protein) family to its receptor on the surface of a target cell, which contributes to the progression of the heart over time.

The aggregation, arrangement and bonding together of a set of components to form a TCR signalosome.

The disaggregation of an adherens junction into its constituent components. An adherens junction is a cell-cell junction composed of the epithelial cadherin-catenin complex at which the cytoplasmic face of the plasma membrane is attached to actin filaments.

Any process that modulates the frequency, rate or extent of anterograde synaptic vesicle transport.

Any process that modulates the frequency, rate or extent of synaptic vesicle transport.

A multi-protein complex containing at least the T-cell receptor complex and the LAT (linker for activation of T cells) scaffold protein. Also contains a variety of signaling proteins including co-receptors, kinases, phosphatases and adaptors such as CD8. Connects events on the plasma membrane to distal signaling cascades to ultimately modulate T cell biology.

The series of molecular signals initiated by binding of a Wnt protein to a frizzled family receptor on the surface of the target cell, followed by propagation of the signal via beta-catenin, and ending with a change in transcription of target genes involved in cardiac neural crest cell differentiation.

The series of molecular signals initiated by binding of a Wnt protein to a frizzled family receptor on the surface of the target cell, followed by propagation of the signal via beta-catenin, and ending with a change in transcription of target genes that contributes to the progression of the heart over time. In this pathway, the activated receptor signals via downstream effectors that result in the inhibition of beta-catenin phosphorylation, thereby preventing degradation of beta-catenin. Stabilized beta-catenin can then accumulate and travel to the nucleus to trigger changes in transcription of target genes.

Any process that stops, prevents or reduces the frequency, rate or extent of phosphatidate phosphatase activity.

Any process that stops, prevents, or reduces the frequency, rate or extent of the torso signaling pathway.

Any process that modulates the frequency, rate or extent of the torso signaling pathway.

The series of molecular signals initiated by binding of a Wnt protein to a frizzled family receptor on the surface of the target cell, followed by propagation of the signal via beta-catenin, and ending with a change in transcription of target genes. In this pathway, the activated receptor signals via downstream effectors that result in the inhibition of beta-catenin phosphorylation, thereby preventing degradation of beta-catenin and contributing to cardiac muscle cell fate commitment. Stabilized beta-catenin can then accumulate and travel to the nucleus to trigger changes in transcription of target genes.

Any process that activates or increases the frequency, rate or extent of anterograde synaptic vesicle transport.

Any process that activates or increases the frequency, rate or extent of synaptic vesicle transport.

Any process that activates or increases the frequency, rate or extent of the torso signaling pathway.

Any process that stops, prevents or reduces the frequency, rate or extent of anterograde synaptic vesicle transport.

Any process that stops, prevents or reduces the frequency, rate or extent of synaptic vesicle transport.

The canonical Wnt signaling pathway that contributes to an expansion of the population of cardiac muscle cells.