The chemical reactions and pathways resulting in the formation of phylloquinone, vitamin K1, a quinone-derived compound synthesized by green plants.

The chemical reactions and pathways resulting in the formation of any of the forms of vitamin K, quinone-derived vitamins which are involved in the synthesis of blood-clotting factors in mammals.

Any process that activates or increases the frequency, rate or extent of interleukin-34 production.

Any process that modulates the frequency, rate or extent of interleukin-34 production.

Any process that stops, prevents or reduces the frequency, rate or extent of interleukin-34 production.

Any process that stops, prevents or reduces the frequency, rate or extent of interleukin-17A production.

Any process that modulates the frequency, rate or extent of interleukin-17A production.

Any process that activates or increases the frequency, rate or extent of interleukin-17A production.

The appearance of interleukin-34 due to biosynthesis or secretion following a cellular stimulus, resulting in an increase in its intracellular or extracellular levels.

The chemical reactions and pathways resulting in the breakdown of biotin, cis-tetrahydro-2-oxothieno(3,4-d)imidazoline-4-valeric acid.

The chemical reactions and pathways resulting in the breakdown of cobalamin (vitamin B12), a water-soluble vitamin characterized by possession of a corrin nucleus containing a cobalt atom.

The chemical reactions and pathways resulting in the breakdown of menaquinones, any of the quinone-derived compounds synthesized by intestinal bacteria. Structurally, menaquinones consist of a methylated naphthoquinone ring structure and side chains composed of a variable number of unsaturated isoprenoid residues. Menaquinones have vitamin K activity and are known as vitamin K2.

Catalysis of the reaction: sphingosine 1-phosphate + H2O = sphingosine + phosphate.

The series of molecular signals mediated by the detection of gibberellic acid and not dependent on the coupling of the alpha subunit of G proteins to the hormone receptors.

Any process that stops, prevents or reduces the frequency, rate or extent of CD86 biosynthetic process.